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Many of the Carisoprodol overdoses have occurred in the setting of multiple drug overdoses including drugs of abuse, illegal drugs, and alcohol. The effects of an overdose of Carisoprodol and other CNS depressants e. Fatal accidental and non-accidental overdoses of Carisoprodol have been reported alone or in combination with CNS depressants.
Basic life support measures should be instituted as dictated by the clinical presentation of the Carisoprodol overdose. Vomiting should not be induced because of the risk of CNS and respiratory depression, and subsequent aspiration.
Circulatory support should be administered with volume infusion and vasopressor agents if needed. Seizures should be treated with intravenous benzodiazepines and the reoccurrence of seizures may be treated with phenobarbital. In cases of severe CNS depression, airway protective reflexes may be compromised and tracheal intubation should be considered for airway protection and respiratory support.
For decontamination in cases of severe toxicity, activated charcoal should be considered in a hospital setting in patients with large overdoses who present early and are not demonstrating CNS depression and can protect their airway. For more information on the management of an overdose of Carisoprodol, contact a Poison Control Center.
Carisoprodol Description Carisoprodol tablets, USP are available as mg and mg round, white tablets. Carisoprodol USP is a white, crystalline powder, having a mild, characteristic odor and a bitter taste.
It is slightly soluble in water; freely soluble in alcohol, in chloroform, and in acetone; and its solubility is practically independent of pH. Carisoprodol is present as a racemic mixture.
Chemically, Carisoprodol is N-isopropylmethylpropyl-1,3-propanediol dicarbamate and the molecular formula is C12H24N2O4, with a molecular weight of The structural formula is: Carisoprodol - Clinical Pharmacology Mechanism of Action The mechanism of action of Carisoprodol in relieving discomfort associated with acute painful musculoskeletal conditions has not been clearly identified.
In animal studies, muscle relaxation induced by Carisoprodol is associated with altered interneuronal activity in the spinal cord and in the descending reticular formation of the brain. Pharmacodynamics Carisoprodol is a centrally acting skeletal muscle relaxant that does not directly relax skeletal muscles.
A metabolite of Carisoprodol, meprobamate, has anxiolytic and sedative properties. The degree to which these properties of meprobamate contribute to the safety and efficacy of Carisoprodol is unknown. The exposure of Carisoprodol and meprobamate was dose proportional between the mg and mg doses.
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